Autophagy and senescence are activated mechanisms in idiopathic or autoimmunity caused usual interstitial pneumonia

H. Popper (Graz, Austria), E. Stacher-Priehse (Gauting, Germany), L. Brcic (Graz, Austria), S. Eidsenhammer (Graz, Austria), F. Gallob (Graz, Austria), F. Rampp (Munich, Germany), A. Nerlich (Munich, Germany)

Source: International Congress 2019 – Translational aspects of idiopathic pulmonary fibrosis
Session: Translational aspects of idiopathic pulmonary fibrosis
Session type: Poster Discussion
Number: 5379
Disease area: Interstitial lung diseases

Congress or journal article abstractE-poster

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Citations should be made in the following way:
H. Popper (Graz, Austria), E. Stacher-Priehse (Gauting, Germany), L. Brcic (Graz, Austria), S. Eidsenhammer (Graz, Austria), F. Gallob (Graz, Austria), F. Rampp (Munich, Germany), A. Nerlich (Munich, Germany). Autophagy and senescence are activated mechanisms in idiopathic or autoimmunity caused usual interstitial pneumonia. 5379

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